Dr. Weyrich's Naturopathic Functional Medicine Notebook is a collection of information on topics of interest
to Dr. Weyrich that may be of interest to the world wide audience.
Due to limitations of time, not all information that Dr. Weyrich knows or would like to further research is
Dr. Weyrich welcomes financial contributions to support specific research topics, as well as copies of
non-free access journal articles for him to review on a topic.
Constructive criticism is also welcome.
Overview of Helicobacter pylori
is the bacteria that causes most cases of
gastrointestinal ulcers. People with low stomach acidity
) are particularly
at risk. Paradoxically, part of the standard treatment for stomach ulcers
is the use of drugs that reduce stomach acid (antacids). While antacid
treatment can relieve the burning symptoms caused by acid irritating the
ulcerated stomach lining, unless an effective antibacterial treatment
regimen is mounted simultaneously, the underlying
infection will be made worse.
Patients with ulcers due to Helicobacter pylori should recover
with proper treatment and should not need to take antacid drugs for
long periods of time. Failure of treatment of Helicobacter pylori
raises concern that the initial diagnosis was incorrect, and additional
diagnostic procedures are warranted to rule out more serious diseases.
Pathophysiology of Helicobacter pylori
grows in the mucosal lining the stomach, where
it is protected from the gastric acid by the mucosa. In addition,
has an enzyme called "urease" that converts
urea into ammonia, which neutralizes stomach acid in its vicinity.
The gastric mucosa also shields the Helicobacter pylori from
the body's immune system. However, the immune system recognizes the
infection and mounts an inflammatory response. This inflammatory response
is largely responsible for the formation of the painful ulcers.
ICD-9 Codes related to Helicobacter pylori
|531.xx||Gastric ulcer|| |
References for Helicobacter pylori